Haemorrhoids
GMJ News knowledge hub · last reviewed September 2026 · Georgian Medical Journal
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Haemorrhoids — symptomatic enlargement and distal displacement of the normal anal cushions — are among the most common anorectal conditions worldwide, affecting an estimated 4-5% of adults symptomatically with far higher rates on examination, and are strongly associated with constipation, prolonged straining, low dietary fibre, pregnancy and prolonged sitting (WHO). The single most important clinical principle is that rectal bleeding must never be attributed to haemorrhoids without excluding colorectal malignancy in patients over 40, those with alarm features (weight loss, altered bowel habit, iron deficiency anaemia, family history) or those whose bleeding does not resolve with treatment — misattribution of cancer bleeding to haemorrhoids is a recurrent and well-documented cause of delayed colorectal cancer diagnosis and successful medicolegal claims.
Key messages
NEVER attribute rectal bleeding to haemorrhoids without excluding cancer
The single most important clinical rule. Rectal bleeding must not be attributed to haemorrhoids in: any patient over 40-50 (threshold varies by guideline and local colorectal cancer incidence); anyone with alarm features (unintentional weight loss, altered bowel habit, iron deficiency anaemia, abdominal or rectal mass, family history of colorectal cancer or IBD); or anyone whose bleeding persists despite treatment. Misattribution of colorectal cancer bleeding to visible haemorrhoids is a well-documented, recurrent cause of delayed cancer diagnosis and of successful medicolegal claims. The presence of haemorrhoids on examination does not exclude a synchronous proximal cancer.
Goligher grading determines treatment
Grade I: bleed but do not prolapse — conservative management (fibre, fluids, avoid straining) ± topical agents. Grade II: prolapse on straining, reduce spontaneously — conservative + office procedures (rubber band ligation first-line). Grade III: prolapse and require manual reduction — rubber band ligation or surgery. Grade IV: irreducibly prolapsed — surgery (haemorrhoidectomy). External haemorrhoids and thrombosed external haemorrhoids are graded separately and managed differently.
Rubber band ligation is the most effective office procedure
Rubber band ligation (RBL): bands applied to the haemorrhoidal pedicle ABOVE the dentate line (insensate — critical point: banding below the dentate line causes severe pain and is a technical error). Most effective non-surgical intervention for grade I-III internal haemorrhoids; superior to sclerotherapy and infrared coagulation in meta-analysis; can be repeated. Complications: pain (usually mild, from banding too low); delayed bleeding at 7-10 days when the band separates (occasionally significant); vasovagal episodes; and rarely, pelvic sepsis (a surgical emergency — presenting with severe pain, fever and urinary retention, requiring immediate assessment). Avoid RBL in anticoagulated patients and in immunosuppression.
Thrombosed external haemorrhoid — the 72-hour window
An acutely thrombosed external haemorrhoid presents as a sudden, exquisitely painful, tense purple perianal lump. If the patient presents WITHIN 72 hours of onset (pain at its peak), excision of the thrombosed haemorrhoid under local anaesthetic gives immediate and dramatic relief and reduces recurrence — this is excision, not simple incision and clot expression, which has higher recurrence. If the patient presents AFTER 72 hours (pain already improving as the clot organises), conservative management is appropriate: analgesia, stool softeners, sitz baths — the lesion resolves spontaneously over 1-2 weeks, often leaving a skin tag.
Anal fissure is the key differential — pain, not painless bleeding
Haemorrhoids characteristically cause PAINLESS bright red bleeding (blood on paper, dripping into the pan, coating rather than mixed into the stool). Severe pain during and after defaecation, often described as passing broken glass, with a small amount of bright blood, indicates ANAL FISSURE — a completely different condition. Fissure treatment: high-fibre diet, stool softeners, sitz baths, and topical vasodilators to reduce internal anal sphincter hypertonia — glyceryl trinitrate (GTN) 0.4% ointment or diltiazem 2% cream (better tolerated, less headache), for 6-8 weeks. Chronic fissure failing medical therapy: botulinum toxin injection or lateral internal sphincterotomy (highly effective but small risk of incontinence).
Fibre and defaecatory habit are the foundation — and the prevention
Conservative management underpins all treatment: soluble fibre supplementation (ispaghula/psyllium — Cochrane evidence for reduced bleeding and symptoms), adequate fluid, and — critically — behavioural change: do not strain; do not sit on the toilet for prolonged periods (reading, phone use — a major and under-addressed contributor); respond promptly to the call to stool. Toilet posture with the knees elevated above the hips (a footstool) straightens the anorectal angle and reduces straining. These measures both treat existing haemorrhoids and prevent recurrence after any procedure — without them, recurrence rates after banding or surgery are high.
Key statistics
Never assume
rectal bleeding is haemorrhoidal without excluding colorectal cancer in at-risk patients
NICE/ASCRSAbove dentate
rubber band ligation must be applied above the dentate line — below causes severe pain
ASCRS72 hours
window for excision of a thrombosed external haemorrhoid — after this, manage conservatively
ASCRSPainful = fissure
severe pain on defaecation indicates anal fissure, not haemorrhoids — different treatment
ASCRS/ACPGBIDiltiazem 2%
topical — as effective as GTN for anal fissure with substantially fewer headaches
CochraneHaemorrhoid treatment by Goligher grade (ASCRS guidelines)
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Related health topics
Colorectal cancer (must exclude)Constipation and strainingIBD (bleeding differential)Haemorrhoids in pregnancyPelvic floor and continenceIron deficiency from chronic bleeding
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